Published Online
on November 5, 2007

A more recent version of this article appeared on December 1, 2007

This Article Full Text (PDF) All Versions of this Article: 50/6/1009    most recent HYPERTENSIONAHA.107.100032v1 Alert me when this article is cited Alert me if a correction is posted Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Download to citation manager Request Permissions Citing Articles Citing Articles via Google Scholar Google Scholar Articles by Paulis, L. Articles by Simko, F. Search for Related Content PubMed PubMed Citation Articles by Paulis, L. Articles by Simko, F. Related Collections Structure Hypertrophy Remodeling Cardiovascular Pharmacology Oxidant stress Endothelium/vascular type/nitric oxide Related Article

LA419, a Novel Nitric Oxide Donor, Prevents Cardiac Remodeling Via the Endothelial Nitric Oxide Synthase Pathway NO Donors as a Means of Antiremodeling

Ludovit Paulis^* and Fedor Simko

From the Department of Pathophysiology (L.P., F.S.), School of Medicine, Comenius University, Bratislava, Slovak Republic; Institute of Normal and Pathological Physiology (L.P.), Slovak Academy of Sciences, Bratislava, Slovak Republic; and the 3rd Clinic of Medicine (F.S.), School of Medicine, Comenius University, Bratislava, Slovak Republic.

^* To whom correspondence should be addressed. E-mail: ludo{at}lfuk.sk.

Related Article:

LA419, a Novel Nitric Oxide Donor, Prevents Pathological Cardiac Remodeling in Pressure-Overloaded Rats Via Endothelial Nitric Oxide Synthase Pathway Regulation

Gema Ruiz-Hurtado, María Fernández-Velasco, Marisabel Mourelle, and Carmen Delgado
Hypertension 2007 50: 1049-1056. [Abstract] [Full Text] [PDF]