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(Hypertension. 2007;50:595.)
© 2007 American Heart Association, Inc.
In Memoriam |
Wake Forest University School of Medicine, Winston-Salem, NC
Carlos Haya University General Hospital, Malaga, Spain
Hospital Clinico, Barcelona, Spain
Hospital Clinico San Carlos, Madrid, Spain
Hospital Clinico, Valencia, Spain
Complutense University, Madrid, Spain
Hospital 12 de Octubre, Madrid, Spain
An extract of the first 250 words of the full text is provided, because this article has no abstract. |
To paraphrase George Orwell, every person is unique, but some are more unique than others. That is the way we wish to remember Manuel Luque Otero, professor of the Universidad Complutense in Madrid, and director of the Hypertension Division of the San Carlos University Hospital in Madrid, Spain (1981–2007). Born in Extremadura, an autonomous community of Western Spain, through his blood ran his ancestors illustrious genes for conquering the unknown, as this region of the Iberic peninsula was the source of many of the most famous Spanish conquerors and settlers in North and Central America. This lineage was reflected in his avid desire to promote new knowledge, his love for medicine and science, his ceaseless pursuit of excellence, his kindness, and his incredible strength during the last moments of his life.
Professor Manolo Luque Otero was a founding member and past president of the Liga Española para la Lucha contra la Hipertension Arterial, vice president of the Sociedad Española de Hipertension, president of the Sociedad Madrileña de Hipertension, founding member of Inter-American Society of Hypertension, and member of the Council for High Blood Pressure Research of the American Heart Association, the American Society of Hypertension, the International Society of Hypertension, and the European Society of Hypertension. His contributions to the field of clinical hypertension included participation in major international clinical trials, his research in the epidemiology of high blood pressure, the first report on the role of angiotensin-(1-7) in mediating the antihypertensive response to angiotensin-converting enzyme inhibition in essential hypertensive
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